Largely correct. It is more about your body either holding on to body fat or using it for energy. There are various ways you can make the body hold on to fat or release fat. Some are more or less healthy depending on personal context.
Many psychiatric meds cause weight gain, specifically fat gain, by driving up insulin. Some others cause increases in appetite. Others cause a reduction in BMR which with no change in diet will cause more fat gain. Some psychiatric meds made getting enough food hard (I’m looking at your Ritalin) and thus have an opposing effect.
Most of the effects on body fat are mediated by hormones like insulin, grehlin, glucagon, and so on. GLP-1 (Glucagon Like Peptide 1) medications target the receptors for glucagon and activate them in a more sustained way that glucagon does, causing changes in appetite along with every availability. There are other effects as well, thus the side effects, but that is the basic mechanism. We actually don’t understand GLP-1 medications fully at this point so we can expect any current description to change over time, but for now that is basically how they work.
Losing weight is not about calories. It is about hunger. Topic is complicated, too lazy to explain, but mainly thats how ozempic became popular
Largely correct. It is more about your body either holding on to body fat or using it for energy. There are various ways you can make the body hold on to fat or release fat. Some are more or less healthy depending on personal context.
Many psychiatric meds cause weight gain, specifically fat gain, by driving up insulin. Some others cause increases in appetite. Others cause a reduction in BMR which with no change in diet will cause more fat gain. Some psychiatric meds made getting enough food hard (I’m looking at your Ritalin) and thus have an opposing effect.
Most of the effects on body fat are mediated by hormones like insulin, grehlin, glucagon, and so on. GLP-1 (Glucagon Like Peptide 1) medications target the receptors for glucagon and activate them in a more sustained way that glucagon does, causing changes in appetite along with every availability. There are other effects as well, thus the side effects, but that is the basic mechanism. We actually don’t understand GLP-1 medications fully at this point so we can expect any current description to change over time, but for now that is basically how they work.
So I can eat 4000 calories a day and only burn 3000 and lose weight? Tell me more!
I infer they mean that you can’t divorce CICO from patterns in hunger signals.